Adipotide (FTPP) 10 mg Adipotide (FTPP) 10 mg
Peptide Hubs

Adipotide (FTPP) 10 mg

ADIPOTIDE 10 MG INJECTION
Drug Class: Targeted Apoptotic Peptide
Composition:
- Active Substance: Adipotide (FTPP)
- Concentration: 10 mg per vial
Presentation: 2 mL Glass Vial
Form: Lyophilized (Freeze-Dried) Powder
Manufacturer: Peptide Hubs
Tested in Laboratory: View Lab Report

$75.00
Shipping From:
International

Peptide Hubs Adipotide (FTPP) 10 mg: The Revolutionary Targeted Fat Destruction Peptide

While most fat-loss compounds work indirectly through appetite suppression or metabolic enhancement, what if you could target and eliminate fat cells directly? Peptide Hubs presents Adipotide for sale, a groundbreaking research compound that represents a paradigm shift in fat reduction technology. Also known as FTPP (Fat-Targeted Proapoptotic Peptide), our Peptide Hubs FTPP is a 10 mg lyophilized powder that works through a unique mechanism of action: inducing programmed cell death (apoptosis) specifically in white adipose tissue. For researchers seeking the next frontier in physique enhancement, Adipotide offers a direct approach to stubborn fat deposits that resist traditional methods.

What is Adipotide (FTPP)?

Adipotide is a novel, synthetically designed peptide conjugate that functions as a targeted apoptotic agent. Its mechanism is fundamentally different from any other fat-loss compound. It consists of a targeting moiety that binds specifically to two proteins (prohibitin and ANXA2) that are highly expressed on the blood vessels supplying white fat cells. Once bound, it delivers a pro-apoptotic synthetic peptide that triggers programmed cell death in the endothelial cells of these blood vessels. This cuts off the blood supply (nutrition and oxygen) to the fat cells, causing them to shrivel and die. The body's natural cleanup crew (macrophages) then removes the cellular debris. This targeted approach means it can potentially address specific fat deposits that are genetically determined and notoriously difficult to lose.

Lab Test Report

adipotide lab test report

Key Effects and Benefits

Research with Adipotide 10 mg has demonstrated several remarkable effects that are of significant interest to the physique enhancement community:

  • Targeted Fat Cell Apoptosis: The primary and most distinctive effect. Adipotide directly causes the death of white fat cells by destroying their blood supply, leading to a literal reduction in fat cell count in treated areas.
  • Reduction of Stubborn Fat Deposits: This makes it a potential tool for targeting genetically determined fat areas that are unresponsive to diet and exercise, such as lower abdominal fat, love handles, and inner thigh fat.
  • Rapid Onset of Action: Unlike compounds that work slowly over months, changes in fat tissue with Adipotide can be observed relatively quickly in research models, often within a short cycle.
  • No Central Nervous System Stimulation: Since it works locally on fat tissue vasculature, it does not cause jitters, anxiety, or other side effects associated with stimulant-based fat burners.
  • Potential for Body Recomposition: By directly removing fat mass, it can significantly improve body composition ratios without the muscle-wasting catabolism often associated with severe caloric deficits. A study on Adipotide's mechanism demonstrated its efficacy in causing rapid fat loss in primate models through targeted apoptosis.
  • Non-Hormonal Mechanism: Adipotide does not work through hormonal pathways like thyroid or catecholamines, making it a unique tool that can be researched alongside other metabolic agents.

Recommended Adipotide Dosage

Dosing for Adipotide is a critical aspect of research due to its potent and targeted nature. As an advanced research compound, protocols are still being refined. Literature suggests dosages in the range of 0.5 mg to 2.0 mg per day for a limited duration. It is typically reconstituted with bacteriostatic water. A crucial aspect of its administration is that it is often used in a localized, subcutaneous manner, injected directly into the subcutaneous fat layer of the target area (e.g., abdomen). The 10 mg vial provides a substantial supply for a focused research cycle. Extreme caution and consultation with a healthcare professional are mandatory before considering research with this compound.

Optimal Adipotide Cycle and Stacking

Research cycles for Adipotide are typically very short due to its powerful effects and potential for side effects. Cycles of 7 to 21 days are common in literature, followed by a significant break. It can be part of a strategic research stack with other Peptide Hubs products for comprehensive body composition management:

  • With AOD 9604 5 mg: Create a powerful dual-mechanism fat loss stack. While Adipotide destroys existing fat cells, AOD 9604 works to enhance lipolysis and inhibit new fat cell formation, attacking the problem from both ends.
  • With Tesamorelin 5 mg: Combine targeted fat destruction with systemic GH-driven lipolysis. This stack addresses both localized stubborn fat (Adipotide) and visceral fat (Tesamorelin) for a comprehensive approach.
  • With Retatrutide 10 mg: For maximum metabolic impact, pair the direct fat-cell targeting of Adipotide with the powerful triple-agonist (GLP-1, GIP, Glucagon) appetite suppression and metabolic enhancement of Retatrutide.
  • With Cagrilintide 10 mg: Research a powerful appetite and fat-mass suppression combination. Adipotide directly removes fat cells, while Cagrilintide (a long-acting amylin analog) potently suppresses appetite and reduces fat mass through a different pathway.
  • With IGF-1 LR3 1 mg: To support muscle preservation during an aggressive fat-loss phase, the localized fat destruction of Adipotide can be researched alongside the potent anabolic and nutrient-partitioning effects of IGF-1 LR3.

Possible Side Effects and Important Considerations

Adipotide is a powerful research compound with a distinct side effect profile that requires careful consideration. Reported effects in research models include:

  • Reversible Kidney Toxicity: The most significant concern is a reversible, dose-dependent increase in markers of kidney stress. This is why short cycles and careful hydration are paramount.
  • Increased urination and thirst, as the body processes the cellular debris from the destroyed fat tissue.
  • Localized bruising, pain, or irritation at the injection site.
  • Generalized fatigue as the body's resources are diverted to clearing apoptotic cells.
  • Potential for loss of lean mass if nutrition and training are not meticulously maintained, as the body may use muscle for energy during this intensive process.

This is an advanced research chemical and should be treated with extreme caution. Sourcing from a reputable supplier like Peptide Hubs is critical for ensuring product purity and accurate dosing, which is non-negotiable for safety. Comprehensive health monitoring, especially of kidney function, is essential during any research involving Adipotide.

Frequently Asked Questions (FAQs)

Is the fat loss from Adipotide permanent?

In theory, yes, for the specific fat cells that are destroyed. Unlike fat loss from dieting where fat cells simply shrink, Adipotide induces apoptosis, meaning those cells are permanently eliminated. However, remaining fat cells can still hypertrophy (enlarge) if a caloric surplus is maintained post-cycle, and new fat cells can still be formed from preadipocytes. Therefore, permanent results are dependent on maintaining a proper diet and lifestyle after the research cycle.

Why are the cycles for Adipotide so short?

The short cycles are primarily a safety precaution due to the observed, reversible kidney toxicity in research models. Limiting the duration of exposure helps manage this risk and allows the body to recover fully between cycles. The potent effects also mean that longer cycles are not necessary to achieve significant results.

Can Adipotide be used for spot reduction?

Yes, this is one of its most unique potential applications. Because it is often administered via localized subcutaneous injection, researchers can target specific adipose deposits. However, it's important to understand that some systemic distribution will occur, and the compound's affinity for white fat vasculature means it will have effects beyond just the injection site, though the greatest effect is typically local.

How does Adipotide compare to Semaglutide or Tirzepatide for fat loss?

They are completely different. Semaglutide and Tirzepatide are GLP-1 receptor agonists that work primarily by suppressing appetite and slowing gastric emptying, leading to reduced caloric intake. Adipotide does not affect appetite; it directly destroys fat cells by cutting off their blood supply. One works on the brain/gut (Semaglutide/Tirzepatide), while the other works directly on the fat tissue itself (Adipotide). They represent two entirely different classes of fat-loss compounds.

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